William B. Farquhar, Ph.D.
Assistant Professor
University of Delaware
Dept. of Health, Nutrition & Exercise Sciences
Telephone: 302-831-6178
Office: Human Performance Lab Room 143
Lab: Human Performance Lab Room 149A
Research Interests
  • Cardiovascular physiology
  • Exercise physiology
Education
  • Post-Doc, Integrative Physiology, Beth Israel Deaconess Medical Center, Harvard Medical School, MA, 2001
  • Ph.D., Exercise Physiology, Penn State University,1998
  • M.S., Cardiac Rehabilitation & Exercise Science, East Stroudsburg University,1991
  • B.S., Exercise Physiology, East Stroudsburg University,1989
Professional Experience
  • 2002-present, Assistant Professor, University of Delaware
  • 2001-2002, Instructor in Medicine, Harvard Medical School Division on Aging, Boston, MA and Research Assistant Scientist, HRCA Research and Training Institute,
  • 1998-2001, Research Fellow, Department of Neurology, BIDMC, Harvard Medical School, Boston, MA
Selected Recent Publications
  • Farquhar WB, Paul EE, Prettyman AV, and Stillabower ME.  Blood Pressure and Hemodynamic Responses to an Acute Sodium Load in Humans. J Appl Physiol 99: 1545-1551, 2005. 
  • Wenner MM, Prettyman AV, Maser RE, Farquhar WB. Preserved Autonomic Function in Amenorrheic Athletes. J Appl Physiol 101: 590-597, 2006.
  • Farquhar WB, Wenner MM, Delaney EP, Prettyman AV, Stillabower ME. Sympathetic Neural Responses to Increased Osmolality in Humans. Am J Physiol Heart Circ Physiol Epub ahead of print June 9, 2006.
  • Young CN, Stillabower ME, DeSabatino A, Farquhar WB. Venous Smooth Muscle Tone and Responsiveness in Older Adults. J Appl Physiol Epub ahead of print July 13, 2006.
Recent Funding
  • Sympathetic – Osmotic Interactions in Humans, NIH, National Heart, Lung, and Blood Institute R15 HL 074851-01,  2/23/04 – 1/31/07, total award: $226,500.
  • Venous Hemodynamic Function in Older Hypertensive Adults, NIH, National Institute on Aging R03 AG 023836-01A1, 1/01/05 – 12/31/06, total award: $151,000
Current Research

In my lab, we are investigating the mechanisms underlying sodium sensitivity of blood pressure in humans.  Our current focus is on elucidating the role of the sympathetic nervous system in contributing to the sodium-induced increases in blood pressure.